KJ, I've been using the KGF with the hairserum for 3 1/2 weeks. Nothing yet but I've been just applying it on the hairline and in and inch or so. As you know the frontal hairline is notoriously hard to get regrowth but I'm hopeful. I've got 1/2 a bottle left. I'll give a report when my bottle is done.

dave

Hi everyone,

I'm brand new to this site.  I love it.  I have a question about the KGF Hair Serum.   I have fine hair that is not very thick.  It is not thinning, it has always been this way.  I am wondering if the KGF would be helpful for me, or is it only for people whose hair is less thick than it used to be?

Thanks for all the great information.

Susan

Hi Susan,

Many months ago (when I was an SAS newbie!) I did a mix of Sea Kelp, LOTS of Grape Seed Proanthocyanidins, plus Coconut Endosperm and Rosehip Oil.  That's all I had at the time, and I started putting in my hair at night and washing it out in the morning.  I also add Sea Kelp to my shampoos (about 1cc per ounce).

For years, I have lost a rather large amount of hair every day in the shower.  Let's put it this way - if my hubbie took a shower after me, he would growl like Chewbaca from Star Wars when he saw the hair in the drain... nice, huh?!  About a month after I started using the mixes above, I literally stopped losing all but a few hairs each day.  And I haven't even tried Chrysin yet! 

I don't know if the actives can foster new growth (although, the KGF seems to have done so for some people) but they seem to have stopped my hair loss, which makes me very happy! 

Tabetha

How about adding a small amount of caffeine?

T.W. Fischer1,2, U.C. Hipler1, P. Elsner1

1Department of Dermatology and Allergology, Friedrich-

Schiller-University, Jena, 2Department of Dermatology and

Androgenetic alopecia (AGA) is a common problem in men of all ages, starting at the age of 20 and affecting about 50% at the age of 50. The underlying cause is an androgen-dependent miniaturization of genetically predetermined hair follicles, modulated by dihydrotestosterone (DHT) and the expression of androgen receptor (AR). Here, we used ex vivo hair follicles from balding areas of men with AGA and cultivated them in vitro to investigate the effects of testosterone and caffeine, the latter being a promising candidate for hair growth stimulation. Hair follicles from 14 biopsies, taken from the vertex areas from male AGA patients, were cultivated for 120–192 h in the presence of normal William’s E medium (control) or William’s E medium containing different concentrations of testosterone and/or caffeine. The outcome parameters were hair shaft elongation and keratinocyte proliferation assessed by Ki-67 staining of longitudinal hair follicle cryosections. Testosterone which is metabolized by intrafollicular 5--reductase to DHT lead to significant growth suppression at the concentration of 5g/ml. This suppression was neutralized by caffeine in concentrations of 0.001 and 0.005%. Moreover, caffeine alone lead to a significant stimulation of hair follicle growth compared to normal medium. The results were confirmed by proliferation assessment with Ki-67 staining. Androgen-dependent growth inhibition of ex vivo hair follicles from patients suffering from AGA was present in the human hair organ culture model. This model imitates the clinical situation of AGA in vitro and may serve for future studies to screen new substances against androgen-dependent hair loss.